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CureFactor for Tinea capitis


Tinea capitis

Tinea capitis is fungal infection of scalp characterised by scaly lesions with varying degree of inflammation and alopecia(baldness).


Causative fungus

  • Microsporum audouini

How the lesion spreads

  • It commonly spreads through direct contact and use of combs of infected individual.

Clinical Features:

Tinea capitis is of three type:

  • Scaly ringworm.
  • Black dot ringworm(trichophyton).
  • Pustular ringworm(tricophyton microcide).

Common symptoms of tinea capitis

  • Onset is usually sub acute and is accompanied with severe itching.
  • Tinae capitis starts as circuar patches with hair loss.
  • Affected skin looks faint rose pink in case of scaly ringworm,black dot appearance,covered with dry,thin greyish scales.


CureFactor for Carbuncle



Carbuncle is one of the common dermatology disease, categorised under deep seated skin eruptions and is infectious disease commonly caused by bacteria staphylococcal. Carbuncle is multilocular,suppurative and extension of furunculosis into the subcutaneous tissue(skin is divided into epidermis-dermis-subcutaneous tissue) characterised by intense induration and sloughing.


Commonly associated with diabetes mellitus and common incidence more in male.
Clinical Features:

  • Carbuncle tend to be slow in onset,the common location of carbuncle are nape of neck,back,buttocks.


  • Fever and pain of the affected part.

How the lesions of carbuncle looks like

  • Carbuncle starts as painful,indurated swelling with affected area dusky red,very tough,tender and has the tend to increase in size gradually.
  • The centre of the lesion is soft and multiple pustules appear at periphery.
  • Foul smelling pus,ulceration with slow healing and permanent scar remains.


  • Carbuncle can prove fatal especially in elderly and debilitated subjects if not treated early.

General Management:

  • Correct the underlying cause.
  • Control diabetes mellitus.


CureFactor for Furunculosis (boils)


Furunculosis (boils):

Boils are skin eruptions that are acute,painful deep eruptions involving dermis. It is commonly caused by staphylococcal infection of hair follicle with cellulitis(inflammation of cellular or adipose tissue that lies beneath the skin) and central suppuration resulting in complete destruction of hair follicle.

Clinical Features:

  • Onset is sudden,location of boils is usually on the back of neck,axillae,face,buttocks,thighs,forearm.


  • Pain of the affected part,sensation of heat over the affected part.

How the lesion looks

  • Boils start of as small red nodule,usually apears in groups(very rarely it appears singly). Boils spread peripherally(away from the centre) and are deep seated.
  • Within 3-4 days the boils becomes inflammed,with bright red appearance,conical in shape,tender.
  • The skin around the boil is stretched glistering and red.
  • Shedding of hair is commonly seen in boils.
  • In 7-10 days the boil becomes ripe,the centre becomes yellow and ruptures with discharge of pus. Pian and tenderness diminishes.


  • Spontaneous rupture and drainage with favourable prognosis.

General Management:

  • Shave the hairs around the boils to avoid its spread.
  • As soon as pus appears it must be drained.


CureFactor for Folliculitis



Folliculitis is defined is infection and inflammation of hair follicles especially of staphylococcal bacteria.

Clinical Features:

Folliculitis is acute condition which lasts for shorter duration. favourite spots or location where you commonly see folliculitis are beard region,neck,scalp,legs,arms,pubic region,eyelashes.


  • Pain in the affected region along with itching,burning sensation.

How the lesions look

  • Folliculitis is superficial lesion that is it arises within the epidermis not involving dermis. Folliculitis starts as superficial pin headed sized pustules. lesions are discrete. Lesions have a pit in centre where you can see the hair follicle, lesions keep appear on other areas. Pustules rupture pus is discharge. later on like every pustular eruptions skin becomes red and swollen. Folliculitis has the tendency to reappear very often and the onset being sudden.

General Management:

  • Good general hygiene.
  • Keep affected area clean,dry.
  • Involvement of beard region avoid shaving.


CureFactor for Erysipelas



Erysipelas are common dermatology disease and is caused by bacterial infection.
Erysipelas is highly contagious streptococcal infection of skin and mucous membrane characterised by painful,red,well defined lesion with fever and regional lymphadenopathy(lymph node enlargement).

Clinical Features:

Onset-sudden,the location of lesion is usually over the face and extremities. Sudden rise of temperature,headache,malaise,nausea,vomiting and intense pain in affected part.

How the lesion looks

  • The lesion starts as dusky redness of affected part,margins raised,well defined. affected parts are swollen,firm,tender. Lesions are peripherally with regional lymphadenopathy. A fter few days sswelling subsides and skin becomes loose and pale. Usually erysipelas leads to complications like gangrene and septicaemia if left untreated for long time.

Course of Erysipelas

  • Resolves within few days or weeks.

General Management

  • Bed rest during fever.
  • Maintain good general hygiene.
  • Keep affected area clean and dry.
  • Good nutritious diet.


CureFactor for Vertigo



Vertigo is sensation of turning of the individual or his environment caused by disease of the labyrinth(ear) of its central connection. Again vertigo is sign that seems to be due to some problem in the ear.


  1. Peripheral(otological)causes(causes that lie within the ear)
  • Any kind of pathological changes in ear( ear is divided into external ear,middle ear and inner ear).
  • Meniere’s disease.
  1. Central(neurological)causes(causes that lie outside the ear)
  • There are few rare diseases where in vertigo is one of the symptom like migraine,epilepsy,multiple sclerosis,hyperventilation,low cardiac output…etc.

Clinical Features:

The set of symptoms appear to be different for peripheral and neurological(central)vertigo.

  • Usually paroxysmal comes in intervals and suddenly
  • Sever type of vertigo with short duration that is the attack lasts for few seconds to minutes.
  • Tinnitus(ringing of ears),deafness is common,vomiting is marked and absence of other neurological abnormalities.


  • Rarely central vertigo is paroxysmal.
  • Not severe with long duration.
  • Deafness is rare,tinnitus is also rare,vomiting absent,presence of neurological abnormalities.


CureFactor for Syncope


Syncope is not a disease its a symptom that is very commonly experienced by many. Syncope is defined as transient sudden loss of consciousness and postural tone due to acute decrease cerebral blood flow.
Syncope is very commonly seen in school going Children’s(especially girls) because of not having their breakfast in the morning(hypoglycaemia).


  1. Vasovagal syncope – commonly seen in susceptible individuals due to emotional and physical stimuli like fright,pain,exhaustion,hot atmosphere,long period of standing in one position.
  2. Carotid sinus syndrome – commonly seen in elderly individual who wear tight collar shirts and on moving neck either to left or right brings on syncope(CUREFACTOR: avoid wearing tight collar shirts).
  3. Cough syncope – this is commonly seen in an individual who suffers from chronic cough,especially paroxymal cough with sudden cough brings on syncope .
  4. Micturition syncope – commonly occurs in male individuals who have urinary tract infection.
  5. Syncope due to metabolic cause
  • Hypoxia – decrease oxygen demand especially seen in high altitudes can bring about syncope.
  • Hypoglycaemia – commonly occurs in an individual who is diabetic patient and also in an individual who has not eating sing long time or prolonged period of syncope can bring about syncope.
  • Hysterical syncope – commonly seen in an hysterical women,which of psychogenic origin.

General Management:

  1. In Vagovagal syncope attacks and syncope in general-immediately lie the individual at once in recumbent position so that blood pressure for cerebral is restored.
  2. Cough syncope- the underlying cause should be treated that is to treat the cough.


CureFactor for Rheumatoid arthritis


Rheumatoid arthritis (RA):
Arthritis means inflammation of joints. In RA,chronic inflammation of joints primarily synovium (knee joint,hip joint,elbow joint) characterised by bilaterally symmetrical polyarthritis(involvement of more then one joint) with positive RA (rheumatoid arthritis) factor.


Exact cause is not known. RA is thought to be an autoimmune disease. Autoimmune disease means body reacts to one won’s antigen cause damage to the organ.
Genetic predisposition is common
Precipitating cause of RA

  1. Physical or emotional stress.
  2. Hormonal disturbances
  • Puberty(transformation from child to an adult).
  • Menopause(menopause means no menstrual bleeding).
  • Commonly seen in womens.

Clinical Features:

  1. First thing that you notice in RA is joint stiffness more marked in morning,pain in the joints,limitation of joint movements.
  2. Fever,malaise,night sweat along with weight loss.
  3. Joint deformities of the affected joint.
  4. RA usually starts with smaller joint involvement affecting bilaterally later on involving the bigger joints.
  5. Joints in RA feels hot on touching the affected joint and tenderness(pain on pressing the affected joint) is present.
  6. Muscular wasting of above and below of the affected joint.
  7. Baker’s cyst(commonly seen with herniation of synovial cavity into the back of the knee causing pain and tenderness of calf).
  8. Common deformities we get to see in RA are:
  • Ulnar deviation(ulnar bone is bone that you see above the wrist joint exactly following the little finger).
  • Swan neck deformity.
  • Z deformity.
  • Boutonniere deformity of fingers.

General Management:

  1. Local rest to affected joint using the splints to prevent deformity.
  2. Start with physiotherapy to avoid contractures.
  3. Regular early morning exercise like simple extension and flexion of affected joint slow and steadily will help relief pain and stiffness.


CureFactor for Osteoporosis


Osteoporosis is condition of decreased quantity and quality of bone resulting in fractures.
pathgenesis of osteoporosis involves the uncoupling of normal balance of bone formation and resorption. Bones are present structurally normal. In case of normal health the amount of formed equals the amount resorbed. In case of osteoporosis either too much bone is resorbed(high turn over) or too little is formed(low turn over).


  1. Risk factors
  2. Nutritional deficiency especially vitamin D.
  3. Post menopausal women.
  4. Cigarette smoking.
  5. Alcohol intake.
  6. Lack of exercise.

Malabsorption syndrome- due to lack of absorption of calcium can cause osteoporosis.
Chronic hyperthyroidism can result in increased bone turn over in osteoporosis.
Weight bearing is necessary for normal bone remodelling,obese persons have lesser chances of getting osteoporosis.


  1. Calcium supplementation is recommended-1500 mg elementary calcium/day.
  2. Vitamin D deficiency is to be treated.
  3. Risk factors of osteoporosis such as smoking alcoholism should be eliminated.


CureFactor for Rickets


Rickets is nutritional deficiency disorder of vitamin D,which is commonly seen in childrens which results in deficient calcification and softening of bones and characterised by bone pains,muscular weakness,deformities and fractures.


  1. Lack of exposure to sunlight.
  2. Low dietary intake of food rich in vitamin D like meat,chicken…etc
  3. Malabsorption syndrome-inability to absorb vitamin D.
  4. Chronic renal failure(impaired calciferol synthesis)-parathormone has its action.
  5. Latrogenic cause.

Clinical Features:

Rickets is disorder which has very insidious onset,in which symptoms and signs take time to appear. commonly childrens are effected and is one of major problem in INDIA. common age group is 4 months to up to 2 years. sex incidence is equal.


  1. Restlessness is the marked feature of rickets which is especially worse at night. Irritability with frequent rolliung of head over the pillow.
  2. Excessive sweating especially on head worse during sleep.
  3. Delayed dentition( dentition means appearance of teeth)
  4. Delay in walking,standing,crawling
  5. Pot belly abdomen is characteristic feature with abdominal weakness look of rickets child
  6. Rickets child has large head with frontal bossing.
  7. Beading of ribs especially 4th,5th and 6th.
  8. Bow of legs and knocking knee.

General Management:

  1. To educate the patient to have diet rich in vitamin D food like egg,milk,meat…etc and also diet rich in calcium. Vitamin D helps calcium absorption so intake of calcium is also must.
  2.  The cheapest means of vitamin D is sunlight,daily exposure at least for 10 mins morning time will be helpful

Adequate exposure to sunlight,eating food like cod liver oil,fish,milk,egg yolk,butter which are rich in vitamin D. Daily requirement of vitamin D is 400IU(International units).
CUREFACTOR:  Avoid intake of cereals it interferes with vitamin D absorption.